Crucial LRDIMM Server Memory Now Available to Support New Intel Xeon Processor E5 Family

Key Messages:

  • Crucial(R) LRDIMMs for Intel(R) Xeon(R) processor E5 family now available
  • New modules support higher memory densities than standard DDR3 RDIMMs, allow users to add more DIMMs per channel, and increase memory bandwidth by up to 35 percent
  • Available in densities up to 32GB and kits up to 96GB

Multimedia Elements:

BOISE, Idaho and GLASGOW, United Kingdom, May 15, 2012 (GLOBE NEWSWIRE) — Crucial, a leading global brand of memory and storage upgrades, today announced the immediate availability of Crucial Load-Reduced DIMM (LRDIMM) server memory in support of the new Intel(R) Xeon(R) processor E5 family. The rise of data center virtualization, cloud computing, and transactional databases has put additional strain on enterprise servers, causing them to struggle to meet user demands. Crucial LRDIMM server memory supports higher memory densities than standard DDR3 RDIMMs, allowing the addition of more DIMMs per channel, and providing flexible, scalable memory that allows applications to perform more effectively. By reducing the electrical loads presented to the CPU, Crucial LRDIMMs deliver up to twice the installed memory capacity for Intel(R) Xeon processor E5 family-based servers, and increase memory bandwidth by up to 35 percent over standard DDR3 RDIMM modules, helping improve overall data center performance and efficiency.

“With adoption of server virtualization, cloud computing, and database applications on the rise, servers and server applications are constantly evolving. As the number of processor cores increases within the data center, server memory must keep pace to enable them to handle heavier workloads,” said Michael Moreland, worldwide product marketing manager, Crucial. “These cores are starving for high-performance memory that can handle increased processing loads both now and in the future, which is why Crucial is committed to supporting advancements in server technologies with compatible, high-quality server memory.”

Crucial DDR3-1333MHz 1.35V LRDIMM server memory is Intel(R)-validated, halogen-free and RoHS compliant. Available in densities up to 32GB and kits up to 96GB, Crucial LRDIMMs are backed by a limited lifetime warranty and are rigorously tested to meet or exceed the high-quality performance specifications customers have come to expect from Crucial. They are available now through select global channel partners, or directly at www.crucial.com.

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About Crucial

Crucial is a leading global brand of Micron Technology, Inc., one of the largest semiconductor manufacturers worldwide. Crucial branded products include industry-leading solid state drives (SSD) and DRAM memory upgrades for desktops, laptops, servers, workstations, and other systems. Crucial products are available worldwide at leading retail and e-tail stores, commercial resellers, and system integrators who can be found at www.crucialproducts.com. For more information or support, visit www.crucial.com

The Crucial logo is available at http://www.globenewswire.com/newsroom/prs/?pkgid=8353

About Micron

Micron Technology, Inc. is one of the world’s leading providers of advanced semiconductor solutions. Through its worldwide operations, Micron manufactures and markets a full range of DRAM, NAND and NOR flash memory, as well as other innovative memory technologies, packaging solutions and semiconductor systems for use in leading-edge computing, consumer, networking, embedded and mobile products. Micron’s common stock is traded on the NASDAQ under the MU symbol. To learn more about Micron Technology, Inc., visit www.micron.com.

(C)2012 Micron Technology, Inc. All rights reserved. Information is subject to change without notice. Crucial and the Crucial logo are trademarks of Micron Technology, Inc. All other brand or product names are trademarks or registered trademarks of their respective holders.

Article source: http://finance.yahoo.com/news/crucial-lrdimm-server-memory-now-130000494.html

Ditching Saturated Fats Could Improve Memory and Cognition

Saturated fats don’t just clog your arteries — they hinder your brain’s effectiveness, too.

bacon-615.jpg

Eating foods that are high in saturated fat — red meat, butter, and other animal products — clog your arteries and increase your risk for heart disease and stroke. Until now, that’s all we thought they did. Now it seems that saturated fats may also be linked to how efficiently our brains work.

In a paper published today in Annals of Neurology, a team of scientists analyzed dietary data from 6,000 women over age 65. Over the course of a four-year monitoring period, women who consumed more saturated fat scored worse on cognitive function tests than those who ate less of the stuff.

What’s more, women who ate healthier types of fat, such as the monounsaturated fats found in olive oil, actually showed improvements in their test results. The findings suggest that swapping one kind of fat for another may not only improve your cardiovascular health, but may also enhance your brain function. That’s particularly important for middle-aged adults who may be at risk for Alzheimer’s, dementia or other degenerative brain disorders.

“The total amount of fat intake did not really matter, but the type of fat did,” said Olivia Okereke, the study’s lead researcher. “Substituting in the good fat in place of the bad fat is a fairly simple dietary modification that could help prevent decline in memory.”

The study drew data from the Women’s Health Study, a 10-year clinical trial of 40,000 women aged 45 and up — so it looks like the jury’s still out for men.

Article source: http://www.theatlantic.com/health/archive/2012/05/ditching-saturated-fats-could-improve-memory-and-cognition/257386/

How exercise improves learning and memory

Washington, May 19 (ANI): The benefits of exercise on the brain have been revealed in many previous studies.

Exercise clears the mind. It gets the blood pumping and more oxygen is delivered to the brain. This is familiar territory, but Dartmouth’s David Bucci thinks there is much more going on.

“In the last several years there have been data suggesting that neurobiological changes are happening-[there are] very brain-specific mechanisms at work here,” said Bucci, an associate professor in the Department of Psychological and Brain Sciences.

From his studies, Bucci and his collaborators have revealed important new findings:

The effects of exercise are different on memory as well as on the brain, depending on whether the exerciser is an adolescent or an adult.

A gene has been identified which seems to mediate the degree to which exercise has a beneficial effect. This has implications for the potential use of exercise as an intervention for mental illness.

Bucci began his pursuit of the link between exercise and memory with attention deficit hyperactivity disorder (ADHD), one of the most common childhood psychological disorders. Bucci is concerned that the treatment of choice seems to be medication.

“The notion of pumping children full of psycho-stimulants at an early age is troublesome. We frankly don’t know the long-term effects of administering drugs at an early age-drugs that affect the brain-so looking for alternative therapies is clearly important,” Bucci cautions.

Anecdotal evidence from colleagues at the University of Vermont started Bucci down the track of ADHD. Based on observations of ADHD children in Vermont summer camps, athletes or team sports players were found to respond better to behavioural interventions than more sedentary children. While systematic empirical data is lacking, this association of exercise with a reduction of characteristic ADHD behaviours was persuasive enough for Bucci.

Coupled with his interest in learning and memory and their underlying brain functions, Bucci and teams of graduate and undergraduate students embarked upon a project of scientific inquiry, investigating the potential connection between exercise and brain function.

Bucci is quick to point out that “the teams of both graduate and undergraduates are responsible for all this work, certainly not just me.” Michael Hopkins, a graduate student at the time, is first author on the papers.

Early on, laboratory rats that exhibit ADHD-like behaviour demonstrated that exercise was able to reduce the extent of these behaviours. The researchers also found that exercise was more beneficial for female rats than males, similar to how it differentially affects male and female children with ADHD.

Moving forward, they investigated a mechanism through which exercise seems to improve learning and memory. This is “brain derived neurotrophic factor” (BDNF) and it is involved in growth of the developing brain. The degree of BDNF expression in exercising rats correlated positively with improved memory, and exercising as an adolescent had longer lasting effects compared to the same duration of exercise, but done as an adult.

“The implication is that exercising during development, as your brain is growing, is changing the brain in concert with normal developmental changes, resulting in your having more permanent wiring of the brain in support of things like learning and memory. It seems important to [exercise] early in life,” stated Bucci.

Bucci’s latest paper was a move to take the studies of exercise and memory in rats and apply them to humans. The subjects in this new study were Dartmouth undergraduates and individuals recruited from the Hanover community.

Bucci said that, “the really interesting finding was that, depending on the person’s genotype for that trophic factor [BDNF], they either did or did not reap the benefits of exercise on learning and memory. This could mean that you may be able to predict which ADHD child, if we genotype them and look at their DNA, would respond to exercise as a treatment and which ones wouldn’t.”

Bucci concluded that the notion that exercise is good for health including mental health is not a huge surprise.

“The interesting question in terms of mental health and cognitive function is how exercise affects mental function and the brain.” This is the question Bucci, his colleagues, and students continue to pursue.

They published papers documenting their results, with the most recent now available in the online version of the journal Neuroscience . (ANI)

Article source: http://in.news.yahoo.com/exercise-improves-learning-memory-051611467.html

HP certifies Hypercloud DDR3 memory modules for Proliant servers

Sony Vaio S Series hands on [Video]


A Sony product specialist shows us the 13.3in model, the S13

Article source: http://feeds.theinquirer.net/c/554/f/7127/s/1f6a70a5/l/0L0Stheinquirer0Bnet0Cinquirer0Cnews0C21752320Chp0Ecertifies0Ehypercloud0Eddr30Ememory0Emodules0Eproliant0Eservers0DWT0Brss0If0FHome0GWT0Brss0Ia0FHP0Kcertifies0KHypercloud0KDDR30Kmemory0Kmodules0Kfor0KProliant0Kservers/story01.htm

Ditching Saturated Fats Could Improve Memory and Cognition

Saturated fats don’t just clog your arteries — they hinder your brain’s effectiveness, too.

bacon-615.jpg

Eating foods that are high in saturated fat — red meat, butter, and other animal products — clog your arteries and increase your risk for heart disease and stroke. Until now, that’s all we thought they did. Now it seems that saturated fats may also be linked to how efficiently our brains work.

In a paper published today in Annals of Neurology, a team of scientists analyzed dietary data from 6,000 women over age 65. Over the course of a four-year monitoring period, women who consumed more saturated fat scored worse on cognitive function tests than those who ate less of the stuff.

What’s more, women who ate healthier types of fat, such as the monounsaturated fats found in olive oil, actually showed improvements in their test results. The findings suggest that swapping one kind of fat for another may not only improve your cardiovascular health, but may also enhance your brain function. That’s particularly important for middle-aged adults who may be at risk for Alzheimer’s, dementia or other degenerative brain disorders.

“The total amount of fat intake did not really matter, but the type of fat did,” said Olivia Okereke, the study’s lead researcher. “Substituting in the good fat in place of the bad fat is a fairly simple dietary modification that could help prevent decline in memory.”

The study drew data from the Women’s Health Study, a 10-year clinical trial of 40,000 women aged 45 and up — so it looks like the jury’s still out for men.

Article source: http://theatlantic.feedsportal.com/c/34375/f/625830/s/1f7bf956/l/0L0Stheatlantic0N0Chealth0Carchive0C20A120C0A50Cditching0Esaturated0Efats0Ecould0Eimprove0Ememory0Eand0Ecognition0C2573860C/story01.htm

Memory Improvement Soars to New Heights, Following Launch of Ultimate Memory …

Providing a structured and proven approach to memory improvement, Ultimate Memory™ announces its launch to the US market.

New York City, New York (PRWEB) May 17, 2012

Following huge success in the South Pacific, Ultimate Memory™ announces its launch within the United States.

The unique memory improvement software has already helped thousands of people around the world train their brains into tip-top shape. The expansion of the company into the U.S market is poised to bring a positive change to the self-improvement software sector.

“At some point in their lives, everyone wishes for an improved memory” says Marc Slater, Managing Director of the company behind Ultimate Memory™.

Continuing, “Our software asks for only ten minutes of attention each day, and provides a very structured approach to improving memory within a short space of time.”

Thousands of people have already purchased the software for use in the home, school or workplace. Containing dozens of fun and engaging activities, Slater and his team believe they have developed a tool that the world was waiting for.

“It’s something that everyone can use, no matter what stage of life they are in and no matter how good their existing memory is. All of us have room for improvement and Ultimate Memory™ allows people to take themselves to the next level at a pace which suits them” Slater adds.

The team is diligently working to improve and update on their offering. Rumours suggest that a big update to the software may be on the horizon, although Slater has made no official announcement to date.

“All I can say is that we don’t plan to give people just one opportunity to improve their memory. As new research comes to light, we’ll act accordingly to ensure that there is always a challenge for brains around the world to tackle” he says.

The software is available online and boasts an impressive guarantee. Dozens of users have already submitted testimonials, providing an impressive marketing platform from which the product was able to launch.

For more information, please visit: http://www.ultimatememory.com

******

About Ultimate Memory™

Ultimate Memory™ is the market’s leading memory improvement software package.

Scientifically proven to improve memory with as little as ten minute’s user per day, Ultimate Memory™ is also backed by an iron-clad guarantee.

Containing many innovative strategies, tutorials, tips and exercises, users benefit from a myriad of varied ways to learn.

For the original version on PRWeb visit: http://www.prweb.com/releases/prweb2012/5/prweb9512234.htm

Article source: http://www.timesunion.com/business/press-releases/article/Memory-Improvement-Soars-to-New-Heights-3565122.php

exercise improves learning and memory

How exercise improves learning and memory Washington: The benefits of exercise on the brain have been revealed in many previous studies.

Exercise clears the mind. It gets the blood pumping and more oxygen is delivered to the brain. This is familiar territory, but Dartmouth’s David Bucci thinks there is much more going on.

“In the last several years there have been data suggesting that neurobiological changes are happening—[there are] very brain-specific mechanisms at work here,” said Bucci, an associate professor in the Department of Psychological and Brain Sciences.

From his studies, Bucci and his collaborators have revealed important new findings:

The effects of exercise are different on memory as well as on the brain, depending on whether the exerciser is an adolescent or an adult.

A gene has been identified which seems to mediate the degree to which exercise has a beneficial effect. This has implications for the potential use of exercise as an intervention for mental illness.

Bucci began his pursuit of the link between exercise and memory with attention deficit hyperactivity disorder (ADHD), one of the most common childhood psychological disorders. Bucci is concerned that the treatment of choice seems to be medication.

“The notion of pumping children full of psycho-stimulants at an early age is troublesome. We frankly don’t know the long-term effects of administering drugs at an early age—drugs that affect the brain—so looking for alternative therapies is clearly important,” Bucci cautions.

Anecdotal evidence from colleagues at the University of Vermont started Bucci down the track of ADHD. Based on observations of ADHD children in Vermont summer camps, athletes or team sports players were found to respond better to behavioural interventions than more sedentary children. While systematic empirical data is lacking, this association of exercise with a reduction of characteristic ADHD behaviours was persuasive enough for Bucci.

Coupled with his interest in learning and memory and their underlying brain functions, Bucci and teams of graduate and undergraduate students embarked upon a project of scientific inquiry, investigating the potential connection between exercise and brain function.

Bucci is quick to point out that “the teams of both graduate and undergraduates are responsible for all this work, certainly not just me.” Michael Hopkins, a graduate student at the time, is first author on the papers.

Early on, laboratory rats that exhibit ADHD-like behaviour demonstrated that exercise was able to reduce the extent of these behaviours. The researchers also found that exercise was more beneficial for female rats than males, similar to how it differentially affects male and female children with ADHD.

Moving forward, they investigated a mechanism through which exercise seems to improve learning and memory. This is “brain derived neurotrophic factor” (BDNF) and it is involved in growth of the developing brain. The degree of BDNF expression in exercising rats correlated positively with improved memory, and exercising as an adolescent had longer lasting effects compared to the same duration of exercise, but done as an adult.

“The implication is that exercising during development, as your brain is growing, is changing the brain in concert with normal developmental changes, resulting in your having more permanent wiring of the brain in support of things like learning and memory. It seems important to [exercise] early in life,” stated Bucci.

Bucci’s latest paper was a move to take the studies of exercise and memory in rats and apply them to humans. The subjects in this new study were Dartmouth undergraduates and individuals recruited from the Hanover community.

Bucci said that, “the really interesting finding was that, depending on the person’s genotype for that trophic factor [BDNF], they either did or did not reap the benefits of exercise on learning and memory. This could mean that you may be able to predict which ADHD child, if we genotype them and look at their DNA, would respond to exercise as a treatment and which ones wouldn`t.”

Bucci concluded that the notion that exercise is good for health including mental health is not a huge surprise.

“The interesting question in terms of mental health and cognitive function is how exercise affects mental function and the brain.” This is the question Bucci, his colleagues, and students continue to pursue.

They published papers documenting their results, with the most recent now available in the online version of the journal Neuroscience .

ANI

Article source: http://zeenews.india.com/news/health/fitness/exercise-improves-learning-and-memory_17046.html

Ditching Saturated Fats Could Improve Memory and Cognition

Saturated fats don’t just clog your arteries — they hinder your brain’s effectiveness, too.

bacon-615.jpg

Eating foods that are high in saturated fat — red meat, butter, and other animal products — clog your arteries and increase your risk for heart disease and stroke. Until now, that’s all we thought they did. Now it seems that saturated fats may also be linked to how efficiently our brains work.

In a paper published today in Annals of Neurology, a team of scientists analyzed dietary data from 6,000 women over age 65. Over the course of a four-year monitoring period, women who consumed more saturated fat scored worse on cognitive function tests than those who ate less of the stuff.

What’s more, women who ate healthier types of fat, such as the monounsaturated fats found in olive oil, actually showed improvements in their test results. The findings suggest that swapping one kind of fat for another may not only improve your cardiovascular health, but may also enhance your brain function. That’s particularly important for middle-aged adults who may be at risk for Alzheimer’s, dementia or other degenerative brain disorders.

“The total amount of fat intake did not really matter, but the type of fat did,” said Olivia Okereke, the study’s lead researcher. “Substituting in the good fat in place of the bad fat is a fairly simple dietary modification that could help prevent decline in memory.”

The study drew data from the Women’s Health Study, a 10-year clinical trial of 40,000 women aged 45 and up — so it looks like the jury’s still out for men.

Article source: http://www.theatlantic.com/health/archive/2012/05/ditching-saturated-fats-could-improve-memory-and-cognition/257386/

Reducing Brain Activity Improves Memory After Cognitive Decline

Newswise — A study led by a Johns Hopkins neuroscientist and published in the May 10 issue of the journal Neuron suggests a potential new therapeutic approach for improving memory and interrupting disease progression in patients with a form of cognitive impairment that often leads to full-blown Alzheimer’s disease.

The focus of the study was “excess brain activity” commonly associated with conditions that cause mild cognitive decline and memory loss, and are linked to an increased risk of Alzheimer’s. Previously, it had been thought that this neural hyperactivity in the hippocampus was the brain’s attempt to compensate for a weakness in forming new memories. Instead, the team found that this excess activity is contributing to conditions such as amnestic mild cognitive impairment (aMCI), in which patients’ memories are worse than would be expected in healthy people the same age.

“In the case of aMCI, it has been suggested that the increased hippocampal activation may serve a beneficial function by recruiting additional neural ‘resources’ to compensate for those that are lost,” explains lead author Michela Gallagher, the Krieger-Eisenhower Professor of Psychological and Brain Sciences in the Johns Hopkins University’s Krieger School of Arts and Sciences. “However, animal studies have raised the alternative view that this excess activation may be contributing to memory impairment.”

To test how a reduction in that hippocampal activity would affect human patients with aMCI, Gallagher’s team administered a low dose of a drug clinically used to treat epilepsy. The goal was to reduce the test subjects’ activity to levels that were similar to those of healthy, age-matched subjects in a control group. They used functional magnetic resonance imaging both to determine the levels of excess activity, and the reduction of it by way of the drug.

Gallagher and her team found that those subjects who had been treated with an effective dose of the drug did better on a memory task, pointing to the therapeutic potential of reducing this excess activation of the hippocampus in patients with aMCI. These findings in human patients with aMCI are the first to clinically demonstrate that over activity in the hippocampus has no benefit for cognition, and are consistent with Gallagher’s research in an animal model of memory loss: aged rodents.

The findings may have broad clinical implications because increased hippocampal activation occurs not only in patients with aMCI, but also in other conditions of risk, such as familial Alzheimer’s disease (AD).

Research in mouse models of familial AD conducted at the Gladstone Institutes of San Francisco has identified mechanisms of the brain that contribute to abnormal excitatory brain activity, as reported in a paper published in the April 27 issue of the journal Cell. In addition, the results of other studies in mice using the same drug used in aMCI patients were presented at last year’s International Congress on Alzheimer’s disease in Paris, showing both improved memory performance and neuronal function in the hippocampus.

“From both a scientific and clinical perspective, I am thrilled about the consistency of findings obtained in aMCI patients and related animal models,” said Lennart Mucke, director of the Gladstone Institute of Neurological Disease and professor of neurology and neuroscience at the University of California San Francisco.

According to Gallagher, the elevated hippocampal activity observed in conditions that precede AD may be one of the underlying mechanisms contributing to neurodegeneration and memory loss. Studies have found that if patients with aMCI are followed for a number of years, those with the greatest excess activation have the greatest further decline in memory, and are more likely to receive a diagnosis of Alzheimer’s over the next four to six years.

“Apart from a direct role in memory impairment, there is concern that elevated activity in vulnerable neural networks could be causing additional damage and possibly promoting the widespread disease-related degeneration that underlies cognitive decline and the conversion to Alzheimer’s disease,” says Gallagher. “Therefore, reducing the elevated activity in the hippocampus may help to restore memory and protect the brain. It will require a carefully monitored, lengthier clinical trial to determine if that is the case.”

The team that conducted the Johns Hopkins study included Arnold Bakker, Greg Krauss, Marilyn Albert, Carolyn Speck, Lauren Jones, Michael Yassa, Amy Shelton and Susan Bassett. The team also included Craig Stark of the University of California at Irvine.

The research was supported by the National Institutes of Health.

Gallagher is the founder of, and a member of the scientific board of, AgeneBio, a biotechnology company focused on developing treatments for diseases that have an impact on memory, such as amnestic mild cognitive impairment and Alzheimer’s disease. The company is headquartered in Indianapolis. Gallagher owns AgeneBio stock, which is subject to certain restrictions under Johns Hopkins policy. She is entitled to shares of any royalties received by the university on sales of products related to her inventorship of intellectual property. The terms of these arrangements are managed by the university in accordance with its conflict-of-interest policies.

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Article source: http://newswise.com/articles/reducing-brain-activity-improves-memory-after-cognitive-decline?ret=/articles/list&category=medicine&page=1&search[status]=3&search[sort]=date+desc&search[section]=10&search[has_multimedia]=

Overeating Might Increase Risk of Memory Loss, Study Suggests

2012-05-17 16:16:00 – The odds of having mild cognitive impairment more than doubled for those in the highest calorie-consuming group compared to those in the lowest.

In a recent study, researchers found that consuming between 2,100 and 6,000 calories per day may double the risk of memory loss, or mild cognitive impairment (MCI), among people age 70 and older. MCI is in between normal age-related memory loss and early Alzheimer’s disease.

“We observed a dose-response pattern which simply means; the higher the amount of calories consumed each day, the higher the risk of MCI,” said study author Yonas Geda, of the Mayo Clinic in Scottsdale, Arizona.

The study involved 1,233 people between the ages of 70 and 89 who were free of dementia and were divided into three equal groups based on their daily caloric consumption. One group consumed between 600 and 1,525 calories a day, the second

between 1,526 and 2,143 and the third between 2,143 and 6,000.

The odds of having MCI more than doubled for those in the highest calorie-consuming group compared to those in the lowest. The results were identical after adjusting for other factors that can affect risk of memory loss. The middle group showed no significant difference in risk.

“Cutting calories and eating foods that make up a healthy diet may be a simpler way to prevent memory loss as we age,” say boomer generation health experts Dian Griesel, Ph.D., and Tom Griesel, authors of the new books TurboCharged: Accelerate Your Fat Burning Metabolism, Get Lean Fast and Leave Diet and Exercise Rules in the Dust (April 2011, BSH) and The TurboCharged Mind (January 2012, BSH). “Always select foods with the highest nutrition per calorie. Avoid all refined and processed man-made foods and stick with fresh fruits, vegetables, nuts, seeds, tubers and quality sources of protein as delivered by Mother Nature herself.”

SOURCE: turbocharged.us.com/too-many-calories-linked-to-memory-loss/

About TurboCharged:

TurboCharged® is a groundbreaking 8-Step program that defies common weight-loss theories. It successfully delivers body-defining rapid fat loss, accelerates metabolism, and improves health and odds of longevity without gimmicks, supplements or special equipment. The TurboCharged Mind is an excellent companion book to the author’s acclaimed rapid fat loss book, TurboCharged, or perfect as a standalone read. A series of supporting TurboCharged™ hypnosis downloads are available for sale via the book’s website, which offers ideal guided meditations to support and direct self-hypnosis sessions for faster fat loss, greater health, reduced stress, and to quit smoking. For more information, log on to www.turbocharged.us.com or follow us on Facebook ( www.facebook.com/turbochargedUS).

Article source: http://www.pr-inside.com/overeating-might-increase-risk-of-memory-r3187244.htm